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Vestibular Physical Therapy in the
Inpatient Setting
A brief introduction and helpful tips for diagnosis, treatment, and
proper referral
Nathan Dugan, PT, DPT Kalispell Regional Medical Center
January 20, 2016
Objectives
● Overview of the vestibular system
○ Anatomy and physiology
○ Vestibular disorders
● History taking
● Screening
● Oculomotor/vestibular assessment
○ Tests and measures
○ Outcome measures
● BPPV diagnosis and treatment
● Outpatient vestibular therapy
● Practical portion
Vestibular Anatomy and
Physiology
Vestibular Anatomy
Bony Labyrinth
● 3 semicircular canals (SCCs),
cochlea, vestibule (central
chamber)
Membranous Labyrinth
● Suspended within bony labyrinth by
perilymphatic fluid
● Contains 5 sensory organs
○ Membranous portions of
SCCs
○ Otolith organs
■ Utricle
■ Saccule
Vestibular Anatomy
Hair Cells
● Contained in each ampulla and otolith organ, convert displacement of the
head into neural firing
Vestibular Anatomy
Semicircular Canals
● Register angular rotations and velocity of the head
● Anterior, posterior, and horizontal
○ Arranged perpendicularly to each other, form coplanar pairs
● Each coplanar pair forms a push-pull relationship
○ Why is this important?
Vestibular Anatomy
Otolith Organs
● Register forces related to linear acceleration (linear head motion/tilt)
● Saccule
○ Vertical
○ Senses anterior-posterior and occipital-caudal motion
● Utricle
○ Horizontal
○ Senses anterior-posterior and interaural motion
Vestibular Reflexes
Vestibulo-ocular Reflex (VOR)
● Acts to maintain stable vision during head motion
● With head turn to the left
○ Firing rate from hair cells in left increase, right decrease
○ Excitatory impulses transmitted to ipsilateral medial rectus/contralateral lateral rectus
○ Inhibitory impulses transmitted to antagonists
○ If retinal image motion is >2 deg/sec, firing rate is modified - VOR IS TRAINABLE
Vestibulospinal Reflex (VSR)
Vestibulocollic Reflex (VCR)
Cervical Reflexes
Cervico-ocular Reflex (COR)
● Interacts with VOR
● Eye movements driven by neck proprioceptors
● Facilitated when vestibular apparatus is injured, however it is rare that COR
has any clinical significance
Cervicospinal Reflex (CSR)
● Acts when body is rotated on stable head
Cervicocollic Reflex (CCR)
Vestibular Disorders
Typical clinical presentations of common disorders
Vestibular Disorders - Clinical Presentation
Benign Paroxysmal Positional Vertigo (BPPV)
● Vertigo lasting 30 seconds to 2 minutes
○ Disappears even if offending head position
is maintained
○ Usually self-limiting and resolves
spontaneously within 6-12 months
● Brought about by assuming specific head
postures
● Common in elderly, any age group after
mild head trauma, F > M
● Very specific nystagmus patterns
depending on canal involvement
● HEARING LOSS, TINNITUS, AURAL
FULLNESS NOT SEEN WITH BPPV
Vestibular Neuritis
● Sudden onset vertigo lasting 48-72 hours
○ Exacerbated by head movement
○ Associated with horizontal - rotatory
nystagmus, postural imbalance, nausea
○ Normal balance returns in ~6 weeks
● Often preceded by a viral infection of
upper respiratory or GI tract (up to 2
weeks)
● Age of onset: 30-60 years
○ Female peak: 40s
○ Male peak: 60s
Vestibular Disorders - Clinical Presentation
Meniere’s Disease/Endolymphatic Hydrops
● Episodic in nature, over many years
● AURAL FULLNESS, HEARING LOSS,
TINNITUS
○ Accompanied by vertigo, imbalance,
nystagmus, nausea, vomiting
○ Vertigo persists from 30 minutes to 24
hours
○ Patient generally ambulatory within 72
hours
● Hearing generally returns but will
eventually fail to return as disease
progresses
● Usually burns itself out
● Age of onset: 40-60 years
Perilymphatic Fistula/Superior SCC Dehiscence
● Patient generally has a history of trauma,
may report a “pop”
● May lead to episodic vertigo and
sensorineural hearing loss
● Tullio phenomenon
○ Vestibular symptoms caused by auditory
stimuli
● Rest generally alleviates symptoms
○ Sneezing, nose blowing may precipitate
Vestibular Disorders - Clinical Presentation
Vestibular Migraine
● Migraine is a common cause of vertigo
and disequilibrium
● Question about headaches and migraines
to determine a link with vestibular
symptoms
● Greater incidence of motion sickness
associated with migraine
Mal de Debarquement Syndrome
● Defined by prolonged/inappropriate
sensations of movement after exposure to
motion
● Occurs after activity such as boat trip,
airplane travel, train travel, etc
● Persistence of symptoms for >/= 1 month
● Find relief when moving
History Taking
What is important to know?
History Taking
● The MOST IMPORTANT part of the initial evaluation
○ Devote some time to this area
● Clinical symptoms will likely begin to lead you to a
diagnosis
● This is a big part of what separates a good vestibular
therapist from other medical professionals with less
knowledge about the vestibular system
● BPPV is a “catch-all” diagnosis
History Taking
Elements that help with diagnosis
1. Tempo
2. Symptoms
3. Circumstances
History Taking - Tempo
● Acute or chronic (>3 days)?
● FIRST onset of dizziness
○ Sudden or slowly developing?
○ Provoked or spontaneous?
○ Prior illness?
● Average duration of spells
History Taking - Symptoms
● What does dizzy mean?
○ Disequilibrium
○ Lightheadedness
○ Rocking/swaying
○ Motion sickness
○ Nausea/vomiting
○ Oscillopsia
○ Floating/swimming/rocking/spinning
○ Vertigo
History Taking - Circumstance
Under what circumstance does the patient’s dizziness occur?
● With certain movements?
● Spontaneously?
● Exacerbated by head/visual movement?
History Taking - Other Considerations
● Often a psychological component to vestibular disorders
● What medications is the patient taking?
Screening Tests
To rule out non-vestibular causes of vertigo
HINTS1
Head Impulse, Nystagmus, Test of Skew
Indicative of stroke:
INFARCT
Impulse Normal, Fast-phase Alternating, Refixation on Cover Test
HINTS plus Hearing
Head Impulse Test
Video: https://www.youtube.com/watch?
v=fiqAkhYNPRk
Direction-Changing Nystagmus
Video: https://www.youtube.com/watch?
v=0rHNappbYtE
Skew Deviation
Video: https://www.youtube.com/watch?
v=-J170K7VAdA
Vertebral Artery Screening
● VBI often presents with vertigo, nausea, and vomiting
○ Other symptoms are important in history
● Clinical tests - poor sensitivity
○ Vertebral artery compression test (VAT)
■ Performed in supine with full cervical rotation/extension
● What might be wrong with this?
○ Modified VAT (mVAT)
■ Patient is in sitting
● Then rotates head to one side and flexes forward at hips thus extending the
cervical spine
Vestibular
Assessment
Oculomotor/Vestibular
Examination
Oculomotor/Vestibulo-Ocular Examination
● Convergence
● Smooth Pursuit
○ Combine with test for gaze-evoked nystagmus for efficiency
● Saccades
● Spontaneous Nystagmus
● Gaze-Evoked Nystagmus
● Skew Deviation
● VOR Cancellation
VOR Testing
● Head Impulse Test
● Head-Shaking Nystagmus
● Dynamic Visual Acuity
● VOR I/II
Outcome Measures
What measures have been validated in the vestibular disorder
population?
Functional Gait Assessment (FGA)
● Modification of DGI to improve reliability and decrease ceiling effect
● 10 items scored 0-3 for total possible score of 30
● May be performed with or without assistive devices
● Equipment
○ Stop watch
○ 20ft x 12in walking path
○ 9in obstacle (2 shoe boxes)
○ Steps
● Cut-off scores2
○ </= 22/30 predicts falls (older adult data)
■ Sensitivity 85%, Specificity 86%
Timed Up-and-Go (TUG)
● 3m (10ft) course, with patient seated in chair to start, and turn in middle
● Timing begins on “go” and ends when patient sits
○ One practice trial should be given
● In vestibular population, both right and left turns should be tested
● Cut-off scores3
○ >11.1s
■ Sensitivity 80%, Specificity 56%
BPPV Diagnosis and Treatment
BPPV
● The only vestibular diagnosis that you can fix with one treatment
○ Therefore it is a great skill for any physical therapist to have
● Remember
○ Characterized by episodic vertigo precipitated by position changes that last for 30s - 2min and
is fatiguing even if the offending head position is maintained
○ Characteristic nystagmus patterns
● Diagnosis can be difficult without the use of Frenzel/infrared goggles
○ Why?
BPPV Mechanism
● Canalithiasis
○ Debris dislodged from the otoconia becomes freely floating in the endolymph of an SCC
○ Head movement causes movement of the otoconia which in turn causes movement of the
endolymph, thus deflecting the cupula
■ Delay in onset of vertigo/nystagmus up to 40 seconds
■ Vertigo generally rises in intensity to a peak, and then improves rapidly and disappears
● Cupulolithiasis
○ Debris dislodged from the otoconia becomes adhered to the cupula
■ Causes sudden onset vertigo/nystagmus that is non-fatiguing
○ Relatively uncommon
● Treatments for canalithiasis and cupulolithiasis are different
Types of BPPV - Posterior Canal
MOST COMMON - 76% of cases
● Diagnostic Test
○ Dix - Hallpike
● Observed Nystagmus and Canal Involvement (Left Dix-Hallpike Test)
○ Left posterior canal - upbeating and left torsional
○ Left anterior canal - downbeating and left torsional
○ Right anterior canal - downbeating and right torsional
● Treatment
○ Canalith Repositioning Treatment/Epley Maneuver (CRT)
○ Liberatory/Semont Maneuver
○ Brandt-Daroff exercises
Posterior Canal BPPV Nystagmus - Video
Video: https://www.youtube.com/watch?
v=jrp8iPfvP4Y
Types of BPPV - Anterior Canal
13% of cases
● Diagnostic Test
○ Dix - Hallpike
● Observed Nystagmus and Canal Involvement (Left Dix-Hallpike Test)
○ Left posterior canal - upbeating and left torsional
○ Left anterior canal - downbeating and left torsional
○ Right anterior canal - downbeating and right torsional
● Treatment
○ Deep Head-Hanging Maneuver4
○ Canalith Repositioning Treatment/Epley Maneuver (CRT)
○ Liberatory/Semont Maneuver
○ Brandt-Daroff exercises
Anterior Canal BPPV Nystagmus - Video
Video: https://www.youtube.com/watch?
v=qW-tBDU9RRc
Types of BPPV - Horizontal Canal
5% of cases
● Diagnostic Test
○ Roll Test
● Observed Nystagmus and Canal Involvement
○ Geotropic (towards floor)
■ Canalithiasis
○ Apogeotropic (away from floor)
■ Cupulolithiasis
○ Which side is involved?
■ More intense nystagmus/increased symptoms and duration on pathological side
● Treatment
○ Bar-B-Que Roll (fast or slow), Appiani/Casani Maneuvers
○ Forced prolonged positioning
Horizontal Canal BPPV Nystagmus - Video
Video: https://www.youtube.com/watch?
v=MtmkD5rDU0o
BPPV Treatment
Lab Portion
Referring to Outpatient Vestibular Therapy
● When should you refer a patient to outpatient vestibular physical therapy?
○ BPPV
○ Other vestibular disorders
● What is outpatient vestibular physical therapy like?
○ Habituation, adaptation, gaze stabilization, balance training
● How long does it take patients to recover?
Questions?
References
1) Newman-Toker DE, Curthoys IS, Halmagyi GM. Diagnosing stroke in acute vertigo: the HINTS family
of eye movement tests and the future of the “eye ECG.” Semin Neurol 2015;35(5):506-521.
2) Wrisley DM, Kumar NA. Functional gait assessment: concurrent, discriminative, and predictive
validity in community-dwelling older adults. Phys Ther 2010;90(5):761-773.
3) Whitney SL, Marchetti GF. The sensitivity and specificity of the timed up & go and the dynamic gait
index for self-reported falls in persons with vestibular disorders. J Vestib Res 2004;14: 397-409.
4) Yacovino DA, Hain TC, Gualtieri F. New therapeutic maneuver for anterior canal benign paroxysmal
positional vertigo. J Neurol 2009:256(11);1851-1855.
All other slides adapted from:
Herdman SJ. Vestibular Rehabilitation, 3ed. Philadelphia: FA Davis; 2007.

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Vestibular Therapy: Diagnosis and Treatment of BPPV

  • 1. Vestibular Physical Therapy in the Inpatient Setting A brief introduction and helpful tips for diagnosis, treatment, and proper referral Nathan Dugan, PT, DPT Kalispell Regional Medical Center January 20, 2016
  • 2. Objectives ● Overview of the vestibular system ○ Anatomy and physiology ○ Vestibular disorders ● History taking ● Screening ● Oculomotor/vestibular assessment ○ Tests and measures ○ Outcome measures ● BPPV diagnosis and treatment ● Outpatient vestibular therapy ● Practical portion
  • 4. Vestibular Anatomy Bony Labyrinth ● 3 semicircular canals (SCCs), cochlea, vestibule (central chamber) Membranous Labyrinth ● Suspended within bony labyrinth by perilymphatic fluid ● Contains 5 sensory organs ○ Membranous portions of SCCs ○ Otolith organs ■ Utricle ■ Saccule
  • 5. Vestibular Anatomy Hair Cells ● Contained in each ampulla and otolith organ, convert displacement of the head into neural firing
  • 6. Vestibular Anatomy Semicircular Canals ● Register angular rotations and velocity of the head ● Anterior, posterior, and horizontal ○ Arranged perpendicularly to each other, form coplanar pairs ● Each coplanar pair forms a push-pull relationship ○ Why is this important?
  • 7. Vestibular Anatomy Otolith Organs ● Register forces related to linear acceleration (linear head motion/tilt) ● Saccule ○ Vertical ○ Senses anterior-posterior and occipital-caudal motion ● Utricle ○ Horizontal ○ Senses anterior-posterior and interaural motion
  • 8. Vestibular Reflexes Vestibulo-ocular Reflex (VOR) ● Acts to maintain stable vision during head motion ● With head turn to the left ○ Firing rate from hair cells in left increase, right decrease ○ Excitatory impulses transmitted to ipsilateral medial rectus/contralateral lateral rectus ○ Inhibitory impulses transmitted to antagonists ○ If retinal image motion is >2 deg/sec, firing rate is modified - VOR IS TRAINABLE Vestibulospinal Reflex (VSR) Vestibulocollic Reflex (VCR)
  • 9. Cervical Reflexes Cervico-ocular Reflex (COR) ● Interacts with VOR ● Eye movements driven by neck proprioceptors ● Facilitated when vestibular apparatus is injured, however it is rare that COR has any clinical significance Cervicospinal Reflex (CSR) ● Acts when body is rotated on stable head Cervicocollic Reflex (CCR)
  • 10. Vestibular Disorders Typical clinical presentations of common disorders
  • 11. Vestibular Disorders - Clinical Presentation Benign Paroxysmal Positional Vertigo (BPPV) ● Vertigo lasting 30 seconds to 2 minutes ○ Disappears even if offending head position is maintained ○ Usually self-limiting and resolves spontaneously within 6-12 months ● Brought about by assuming specific head postures ● Common in elderly, any age group after mild head trauma, F > M ● Very specific nystagmus patterns depending on canal involvement ● HEARING LOSS, TINNITUS, AURAL FULLNESS NOT SEEN WITH BPPV Vestibular Neuritis ● Sudden onset vertigo lasting 48-72 hours ○ Exacerbated by head movement ○ Associated with horizontal - rotatory nystagmus, postural imbalance, nausea ○ Normal balance returns in ~6 weeks ● Often preceded by a viral infection of upper respiratory or GI tract (up to 2 weeks) ● Age of onset: 30-60 years ○ Female peak: 40s ○ Male peak: 60s
  • 12. Vestibular Disorders - Clinical Presentation Meniere’s Disease/Endolymphatic Hydrops ● Episodic in nature, over many years ● AURAL FULLNESS, HEARING LOSS, TINNITUS ○ Accompanied by vertigo, imbalance, nystagmus, nausea, vomiting ○ Vertigo persists from 30 minutes to 24 hours ○ Patient generally ambulatory within 72 hours ● Hearing generally returns but will eventually fail to return as disease progresses ● Usually burns itself out ● Age of onset: 40-60 years Perilymphatic Fistula/Superior SCC Dehiscence ● Patient generally has a history of trauma, may report a “pop” ● May lead to episodic vertigo and sensorineural hearing loss ● Tullio phenomenon ○ Vestibular symptoms caused by auditory stimuli ● Rest generally alleviates symptoms ○ Sneezing, nose blowing may precipitate
  • 13. Vestibular Disorders - Clinical Presentation Vestibular Migraine ● Migraine is a common cause of vertigo and disequilibrium ● Question about headaches and migraines to determine a link with vestibular symptoms ● Greater incidence of motion sickness associated with migraine Mal de Debarquement Syndrome ● Defined by prolonged/inappropriate sensations of movement after exposure to motion ● Occurs after activity such as boat trip, airplane travel, train travel, etc ● Persistence of symptoms for >/= 1 month ● Find relief when moving
  • 14. History Taking What is important to know?
  • 15. History Taking ● The MOST IMPORTANT part of the initial evaluation ○ Devote some time to this area ● Clinical symptoms will likely begin to lead you to a diagnosis ● This is a big part of what separates a good vestibular therapist from other medical professionals with less knowledge about the vestibular system ● BPPV is a “catch-all” diagnosis
  • 16. History Taking Elements that help with diagnosis 1. Tempo 2. Symptoms 3. Circumstances
  • 17. History Taking - Tempo ● Acute or chronic (>3 days)? ● FIRST onset of dizziness ○ Sudden or slowly developing? ○ Provoked or spontaneous? ○ Prior illness? ● Average duration of spells
  • 18. History Taking - Symptoms ● What does dizzy mean? ○ Disequilibrium ○ Lightheadedness ○ Rocking/swaying ○ Motion sickness ○ Nausea/vomiting ○ Oscillopsia ○ Floating/swimming/rocking/spinning ○ Vertigo
  • 19. History Taking - Circumstance Under what circumstance does the patient’s dizziness occur? ● With certain movements? ● Spontaneously? ● Exacerbated by head/visual movement?
  • 20. History Taking - Other Considerations ● Often a psychological component to vestibular disorders ● What medications is the patient taking?
  • 21. Screening Tests To rule out non-vestibular causes of vertigo
  • 22. HINTS1 Head Impulse, Nystagmus, Test of Skew Indicative of stroke: INFARCT Impulse Normal, Fast-phase Alternating, Refixation on Cover Test HINTS plus Hearing
  • 23. Head Impulse Test Video: https://www.youtube.com/watch? v=fiqAkhYNPRk
  • 26. Vertebral Artery Screening ● VBI often presents with vertigo, nausea, and vomiting ○ Other symptoms are important in history ● Clinical tests - poor sensitivity ○ Vertebral artery compression test (VAT) ■ Performed in supine with full cervical rotation/extension ● What might be wrong with this? ○ Modified VAT (mVAT) ■ Patient is in sitting ● Then rotates head to one side and flexes forward at hips thus extending the cervical spine
  • 29. Oculomotor/Vestibulo-Ocular Examination ● Convergence ● Smooth Pursuit ○ Combine with test for gaze-evoked nystagmus for efficiency ● Saccades ● Spontaneous Nystagmus ● Gaze-Evoked Nystagmus ● Skew Deviation ● VOR Cancellation
  • 30. VOR Testing ● Head Impulse Test ● Head-Shaking Nystagmus ● Dynamic Visual Acuity ● VOR I/II
  • 31. Outcome Measures What measures have been validated in the vestibular disorder population?
  • 32. Functional Gait Assessment (FGA) ● Modification of DGI to improve reliability and decrease ceiling effect ● 10 items scored 0-3 for total possible score of 30 ● May be performed with or without assistive devices ● Equipment ○ Stop watch ○ 20ft x 12in walking path ○ 9in obstacle (2 shoe boxes) ○ Steps ● Cut-off scores2 ○ </= 22/30 predicts falls (older adult data) ■ Sensitivity 85%, Specificity 86%
  • 33. Timed Up-and-Go (TUG) ● 3m (10ft) course, with patient seated in chair to start, and turn in middle ● Timing begins on “go” and ends when patient sits ○ One practice trial should be given ● In vestibular population, both right and left turns should be tested ● Cut-off scores3 ○ >11.1s ■ Sensitivity 80%, Specificity 56%
  • 34. BPPV Diagnosis and Treatment
  • 35. BPPV ● The only vestibular diagnosis that you can fix with one treatment ○ Therefore it is a great skill for any physical therapist to have ● Remember ○ Characterized by episodic vertigo precipitated by position changes that last for 30s - 2min and is fatiguing even if the offending head position is maintained ○ Characteristic nystagmus patterns ● Diagnosis can be difficult without the use of Frenzel/infrared goggles ○ Why?
  • 36. BPPV Mechanism ● Canalithiasis ○ Debris dislodged from the otoconia becomes freely floating in the endolymph of an SCC ○ Head movement causes movement of the otoconia which in turn causes movement of the endolymph, thus deflecting the cupula ■ Delay in onset of vertigo/nystagmus up to 40 seconds ■ Vertigo generally rises in intensity to a peak, and then improves rapidly and disappears ● Cupulolithiasis ○ Debris dislodged from the otoconia becomes adhered to the cupula ■ Causes sudden onset vertigo/nystagmus that is non-fatiguing ○ Relatively uncommon ● Treatments for canalithiasis and cupulolithiasis are different
  • 37. Types of BPPV - Posterior Canal MOST COMMON - 76% of cases ● Diagnostic Test ○ Dix - Hallpike ● Observed Nystagmus and Canal Involvement (Left Dix-Hallpike Test) ○ Left posterior canal - upbeating and left torsional ○ Left anterior canal - downbeating and left torsional ○ Right anterior canal - downbeating and right torsional ● Treatment ○ Canalith Repositioning Treatment/Epley Maneuver (CRT) ○ Liberatory/Semont Maneuver ○ Brandt-Daroff exercises
  • 38. Posterior Canal BPPV Nystagmus - Video Video: https://www.youtube.com/watch? v=jrp8iPfvP4Y
  • 39. Types of BPPV - Anterior Canal 13% of cases ● Diagnostic Test ○ Dix - Hallpike ● Observed Nystagmus and Canal Involvement (Left Dix-Hallpike Test) ○ Left posterior canal - upbeating and left torsional ○ Left anterior canal - downbeating and left torsional ○ Right anterior canal - downbeating and right torsional ● Treatment ○ Deep Head-Hanging Maneuver4 ○ Canalith Repositioning Treatment/Epley Maneuver (CRT) ○ Liberatory/Semont Maneuver ○ Brandt-Daroff exercises
  • 40. Anterior Canal BPPV Nystagmus - Video Video: https://www.youtube.com/watch? v=qW-tBDU9RRc
  • 41. Types of BPPV - Horizontal Canal 5% of cases ● Diagnostic Test ○ Roll Test ● Observed Nystagmus and Canal Involvement ○ Geotropic (towards floor) ■ Canalithiasis ○ Apogeotropic (away from floor) ■ Cupulolithiasis ○ Which side is involved? ■ More intense nystagmus/increased symptoms and duration on pathological side ● Treatment ○ Bar-B-Que Roll (fast or slow), Appiani/Casani Maneuvers ○ Forced prolonged positioning
  • 42. Horizontal Canal BPPV Nystagmus - Video Video: https://www.youtube.com/watch? v=MtmkD5rDU0o
  • 44. Referring to Outpatient Vestibular Therapy ● When should you refer a patient to outpatient vestibular physical therapy? ○ BPPV ○ Other vestibular disorders ● What is outpatient vestibular physical therapy like? ○ Habituation, adaptation, gaze stabilization, balance training ● How long does it take patients to recover?
  • 46. References 1) Newman-Toker DE, Curthoys IS, Halmagyi GM. Diagnosing stroke in acute vertigo: the HINTS family of eye movement tests and the future of the “eye ECG.” Semin Neurol 2015;35(5):506-521. 2) Wrisley DM, Kumar NA. Functional gait assessment: concurrent, discriminative, and predictive validity in community-dwelling older adults. Phys Ther 2010;90(5):761-773. 3) Whitney SL, Marchetti GF. The sensitivity and specificity of the timed up & go and the dynamic gait index for self-reported falls in persons with vestibular disorders. J Vestib Res 2004;14: 397-409. 4) Yacovino DA, Hain TC, Gualtieri F. New therapeutic maneuver for anterior canal benign paroxysmal positional vertigo. J Neurol 2009:256(11);1851-1855. All other slides adapted from: Herdman SJ. Vestibular Rehabilitation, 3ed. Philadelphia: FA Davis; 2007.